LYCOS RETRIEVER 
Herpes: United States
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The immune system is able to destroy active herpes virus particles but the herpes virus has the ability to hide from the immune system in an inactive (or latent) state. Current research suggests that this ability to hide may be achieved via modification to cellular enzyme histone deacetylases (HDACs), namely HDAC1 and HDAC2. [53] Hypothetically, by interfering with the HDAC enzymes' effectiveness, it may be possible to block the virus's ability to hide from the immune system, leading to a complete elimination of the virus by the immune system. Studies on the impact of HDAC inhibitors on viral latency are ongoing in the HIV arena.
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Incidence of genital herpes in the United States has increased 30% over the past 20 years. The largest increase has occurred in Caucasians between the ages of 12 and 19 (5 times more common) and ages 20–29 (twice as common). About 1 million new cases occur each year.
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The second is Simplex Type 2 or (HSV-2) which is responsible for Genital Herpes. This virus in its dormant state hides in the nerves at the base of the spine and outbreaks are usually contained to the genital area.
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About one in five adults in the United States has genital herpes. However, most people don’t know they are infected because their symptoms are too mild to notice or mistaken for another condition.
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The herpes virus establishes a latent state in sensory and autonomic ganglia. Although the frequency and severity of recurrent outbreaks may vary greatly depending upon the individual, physical and/or mental stress have been commonly noted as contributors to an outbreak.
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